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Sep 3, 20151 like761 views

Check out 10 common myths that can impede your Fitness Goal. It may stop your profess. Know the truth behind 10 common fitness myths.

Есть интерес, а есть исследование. Фокус группа - метод социального (маркетингового или др) исследования, со своей историей и практиками. Когда я слышу от умного человека компетентного в своем деле человека, но далекого от исследований, что он провел вчера в баре фокус группу, я начинаю понимать граммар наци Несколько слайдов прояснить разницу между фокус группой и не фокус группой. И да, я сама проводила экспериментальные группы в пиццериях, и без гайда, и со знакомыми - но в порядке эксперимента, таки с аджендой.

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Croup is a common childhood illness characterized by swelling in the airway (larynx, Trachea and bronchi), leading to a distinctive barking cough and difficulty in breathing, often with a high-pitched, noisy sound called stridor when inhaling.  It is also known as laryngotracheobronchitis. THE WESTLEY SCORE: classification of croup severity SCORING SYSTEM:  A total score of < 2 indicates mild croup.  A total score of 3-5 is classifies as moderate croup.  A total score of 6-11 indicates severe croup.  A total score of >12 indicates impending respiratory failure.  The score ranges from 0-17. MANAGEMENT:  Supportive care including intravenous fluids to maintain hydration and oxygen inhalation to relieve hypoxia has to be given.  A single dose of dexamethasone 0.6mg/kg may decrease severity and duration of illness.  Inhalation of epinephrine may decrease the symptoms of stridor and respiratory distress immediately. PREVENTION:  Many cases of croup have been prevented by immunization for influenza and diphtheria.

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Making meaning from hardship is a complex conversation. Many cancer survivors feel the delicate balance between making meaning and the internalized or external pressure that often follows a cancer diagnosis. Questions such as “What now?” are common when treatment ends. Well-meaning friends and family may subtly (or not so subtly) expect us to behave or view the world differently. If figuring out who you are now feels puzzling, join us on Wednesday, December 11th. Together, we will discuss how changes in your identity and perspective are a valid and essential part of this journey. Research has shown us how making meaning after hardship facilitates adjustment and well-being.

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diabetes mcq by NAME ANKUSH GOYAL (1).pdf by Dr Ankush goyal, has 14 slides with 118 views.Diabetes Mellitus: A Comprehensive Overview Introduction Diabetes mellitus is a chronic metabolic disorder characterized by hyperglycemia due to defects in insulin secretion, insulin action, or both. It affects millions of people worldwide and is a major cause of morbidity and mortality due to its associated complications. This document provides an in-depth discussion of the types, pathophysiology, clinical features, diagnosis, management, and complications of diabetes mellitus. Types of Diabetes Mellitus 1. Type 1 Diabetes Mellitus (T1DM) Autoimmune destruction of pancreatic beta cells Absolute insulin deficiency Typically presents in childhood or adolescence Requires lifelong insulin therapy 2. Type 2 Diabetes Mellitus (T2DM) Characterized by insulin resistance and relative insulin deficiency Strong genetic predisposition Associated with obesity and sedentary lifestyle Managed with lifestyle modifications, oral hypoglycemics, and sometimes insulin 3. Gestational Diabetes Mellitus (GDM) Hyperglycemia first recognized during pregnancy Increases risk of complications for both mother and baby Usually resolves postpartum but increases the risk of T2DM later in life 4. Other Specific Types Monogenic diabetes (MODY, neonatal diabetes) Secondary diabetes (due to pancreatic diseases, endocrinopathies, drug-induced, etc.) Pathophysiology Diabetes results from impaired insulin secretion, action, or both, leading to chronic hyperglycemia. The key mechanisms include: Type 1 Diabetes: Autoimmune destruction of beta cells, leading to absolute insulin deficiency. Type 2 Diabetes: Insulin resistance in peripheral tissues and inadequate compensatory insulin secretion by beta cells. GDM: Hormonal changes in pregnancy lead to insulin resistance and beta-cell dysfunction. Clinical Features Symptoms of Hyperglycemia: Polyuria (excessive urination) Polydipsia (excessive thirst) Polyphagia (excessive hunger) Unexplained weight loss Fatigue Blurred vision Complications: Acute: Diabetic ketoacidosis (DKA), hyperosmolar hyperglycemic state (HHS) Chronic: Microvascular (retinopathy, nephropathy, neuropathy) and macrovascular (coronary artery disease, stroke, peripheral artery disease) Diagnosis The diagnosis of diabetes is based on: Fasting Plasma Glucose (FPG) ≥ 126 mg/dL Random Plasma Glucose ≥ 200 mg/dL with symptoms of hyperglycemia 2-hour Plasma Glucose ≥ 200 mg/dL during an OGTT Hemoglobin A1c ≥ 6.5% Management 1. Lifestyle Modifications Healthy diet (low glycemic index, high fiber, reduced saturated fats) Regular physical activity (at least 150 minutes per week) Weight management 2. Pharmacological Therapy Oral Hypoglycemics: Metformin (first-line), sulfonylureas, DPP-4 inhibitors, SGLT2 inhibitors, thiazolidinediones Injectable Therapy: Insulin, GLP-1 receptor agonists Insulin Therapy: Required for T1DM and some cases of T2DM 3. Monitoring and Complication Prevention Regular blood glucose

diabetes mcq by NAME ANKUSH GOYAL (1).pdfDr Ankush goyal

14 slides•118 views

Diabetes Mellitus: A Comprehensive Overview Introduction Diabetes mellitus is a chronic metabolic disorder characterized by hyperglycemia due to defects in insulin secretion, insulin action, or both. It affects millions of people worldwide and is a major cause of morbidity and mortality due to its associated complications. This document provides an in-depth discussion of the types, pathophysiology, clinical features, diagnosis, management, and complications of diabetes mellitus. Types of Diabetes Mellitus 1. Type 1 Diabetes Mellitus (T1DM) Autoimmune destruction of pancreatic beta cells Absolute insulin deficiency Typically presents in childhood or adolescence Requires lifelong insulin therapy 2. Type 2 Diabetes Mellitus (T2DM) Characterized by insulin resistance and relative insulin deficiency Strong genetic predisposition Associated with obesity and sedentary lifestyle Managed with lifestyle modifications, oral hypoglycemics, and sometimes insulin 3. Gestational Diabetes Mellitus (GDM) Hyperglycemia first recognized during pregnancy Increases risk of complications for both mother and baby Usually resolves postpartum but increases the risk of T2DM later in life 4. Other Specific Types Monogenic diabetes (MODY, neonatal diabetes) Secondary diabetes (due to pancreatic diseases, endocrinopathies, drug-induced, etc.) Pathophysiology Diabetes results from impaired insulin secretion, action, or both, leading to chronic hyperglycemia. The key mechanisms include: Type 1 Diabetes: Autoimmune destruction of beta cells, leading to absolute insulin deficiency. Type 2 Diabetes: Insulin resistance in peripheral tissues and inadequate compensatory insulin secretion by beta cells. GDM: Hormonal changes in pregnancy lead to insulin resistance and beta-cell dysfunction. Clinical Features Symptoms of Hyperglycemia: Polyuria (excessive urination) Polydipsia (excessive thirst) Polyphagia (excessive hunger) Unexplained weight loss Fatigue Blurred vision Complications: Acute: Diabetic ketoacidosis (DKA), hyperosmolar hyperglycemic state (HHS) Chronic: Microvascular (retinopathy, nephropathy, neuropathy) and macrovascular (coronary artery disease, stroke, peripheral artery disease) Diagnosis The diagnosis of diabetes is based on: Fasting Plasma Glucose (FPG) ≥ 126 mg/dL Random Plasma Glucose ≥ 200 mg/dL with symptoms of hyperglycemia 2-hour Plasma Glucose ≥ 200 mg/dL during an OGTT Hemoglobin A1c ≥ 6.5% Management 1. Lifestyle Modifications Healthy diet (low glycemic index, high fiber, reduced saturated fats) Regular physical activity (at least 150 minutes per week) Weight management 2. Pharmacological Therapy Oral Hypoglycemics: Metformin (first-line), sulfonylureas, DPP-4 inhibitors, SGLT2 inhibitors, thiazolidinediones Injectable Therapy: Insulin, GLP-1 receptor agonists Insulin Therapy: Required for T1DM and some cases of T2DM 3. Monitoring and Complication Prevention Regular blood glucose

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Growth and development milestone 7 months .pptxAbhishek Verma

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Growth and developed milestone for a 7 months old children Milestone 7 months At seven months, your baby is becoming more active, curious, and expressive! Many can sit up without support, reach for toys, and even start rocking on their hands and knees, a sign that crawling is coming soon. Their grip is improving, so they enjoy grabbing, shaking, and banging objects together. Socially, they recognize their name, respond to familiar voices, and may show separation anxiety when a parent leaves the room. They love interactive games like peekaboo and enjoy babbling sounds like “ba-ba” and “da-da.” Solid foods are now a regular part of their diet, along with breast milk or formula. They may sleep 12–16 hours a day, including two or three naps. Every baby develops at their own pace, so don’t worry if they’re not hitting every milestone at the same time as others. Enjoy this exciting stage of discovery and growth. Discover the essential growth and development milestones of a 7-month-old baby. Learn about physical, cognitive, emotional, and social developments, including motor skills, sensory awareness, speech progress, and feeding habits. Get expert parenting tips, activities, and guidance to support your baby's healthy development. Stay informed with this comprehensive guide Wondering what milestones your 7-month-old should be reaching? This expert guide covers key physical, cognitive, and social developments, from sitting up and babbling to improved hand-eye coordination. Learn about feeding, sleep patterns, and fun activities to support your baby's growth. Get top parenting tips and insights to ensure your little one thrives! #pediatricnursing #Growthanddevelopment #Growthanddevelopmentalmilestones Growth and development ppt Growth and development milestone slideshare ppt

Psychopharmacology questions .pdf by Dr ankush goyal by Dr Ankush goyal, has 17 slides with 124 views.Psychopharmacology: A Comprehensive Overview Introduction Psychopharmacology is the scientific study of the effects of drugs on mood, perception, cognition, and behavior. It involves the interaction of drugs with the nervous system, particularly the brain, to alter mental functions and treat psychiatric disorders. Psychopharmacology integrates principles from neuroscience, pharmacology, psychiatry, and psychology to develop effective treatments for mental illnesses such as depression, schizophrenia, anxiety, and bipolar disorder. History and Development The field of psychopharmacology has its roots in ancient medicine, where herbal remedies were used to treat mental disorders. Modern psychopharmacology began in the 20th century with the discovery of drugs such as chlorpromazine (antipsychotic) and imipramine (antidepressant), revolutionizing psychiatric care. Over time, research has led to the development of more targeted and effective drugs with fewer side effects. Neurotransmitter Systems and Drug Actions Psychotropic drugs exert their effects by modulating neurotransmitter systems in the brain. The major neurotransmitters involved include: Dopamine (DA): Associated with reward, motivation, and movement; implicated in schizophrenia and Parkinson’s disease. Serotonin (5-HT): Regulates mood, appetite, and sleep; targeted by antidepressants. Norepinephrine (NE): Involved in arousal and stress response; plays a role in depression and anxiety. Gamma-Aminobutyric Acid (GABA): The primary inhibitory neurotransmitter; targeted by anxiolytics and sedatives. Glutamate: The primary excitatory neurotransmitter; implicated in schizophrenia and cognitive function. Acetylcholine (ACh): Important for memory and learning; affected in Alzheimer’s disease. Classification of Psychotropic Drugs Psychotropic drugs can be classified based on their therapeutic use: 1. Antidepressants Used to treat depressive disorders, anxiety disorders, and other conditions. Selective Serotonin Reuptake Inhibitors (SSRIs): Fluoxetine, Sertraline, Citalopram Serotonin-Norepinephrine Reuptake Inhibitors (SNRIs): Venlafaxine, Duloxetine Tricyclic Antidepressants (TCAs): Amitriptyline, Imipramine Monoamine Oxidase Inhibitors (MAOIs): Phenelzine, Selegiline Atypical Antidepressants: Bupropion, Mirtazapine 2. Antipsychotics Used to manage schizophrenia, bipolar disorder, and psychotic disorders. Typical (First-Generation) Antipsychotics: Haloperidol, Chlorpromazine Atypical (Second-Generation) Antipsychotics: Risperidone, Olanzapine, Clozapine 3. Mood Stabilizers Used to treat bipolar disorder and mood dysregulation. Lithium: Effective for bipolar disorder Anticonvulsants: Valproate, Lamotrigine, Carbamazepine 4. Anxiolytics and Sedative-Hypnotics Used for anxiety and sleep disorders. Benzodiazepines: Diazepam, Lorazepam, Alprazolam Non-Benzodiazepine Hypnotics: Zolpidem, Eszopiclone Barbiturates: Phenobarbital (rarely used) Beta-Blockers: Propranolol

Psychopharmacology questions .pdf by Dr ankush goyalDr Ankush goyal

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Psychopharmacology: A Comprehensive Overview Introduction Psychopharmacology is the scientific study of the effects of drugs on mood, perception, cognition, and behavior. It involves the interaction of drugs with the nervous system, particularly the brain, to alter mental functions and treat psychiatric disorders. Psychopharmacology integrates principles from neuroscience, pharmacology, psychiatry, and psychology to develop effective treatments for mental illnesses such as depression, schizophrenia, anxiety, and bipolar disorder. History and Development The field of psychopharmacology has its roots in ancient medicine, where herbal remedies were used to treat mental disorders. Modern psychopharmacology began in the 20th century with the discovery of drugs such as chlorpromazine (antipsychotic) and imipramine (antidepressant), revolutionizing psychiatric care. Over time, research has led to the development of more targeted and effective drugs with fewer side effects. Neurotransmitter Systems and Drug Actions Psychotropic drugs exert their effects by modulating neurotransmitter systems in the brain. The major neurotransmitters involved include: Dopamine (DA): Associated with reward, motivation, and movement; implicated in schizophrenia and Parkinson’s disease. Serotonin (5-HT): Regulates mood, appetite, and sleep; targeted by antidepressants. Norepinephrine (NE): Involved in arousal and stress response; plays a role in depression and anxiety. Gamma-Aminobutyric Acid (GABA): The primary inhibitory neurotransmitter; targeted by anxiolytics and sedatives. Glutamate: The primary excitatory neurotransmitter; implicated in schizophrenia and cognitive function. Acetylcholine (ACh): Important for memory and learning; affected in Alzheimer’s disease. Classification of Psychotropic Drugs Psychotropic drugs can be classified based on their therapeutic use: 1. Antidepressants Used to treat depressive disorders, anxiety disorders, and other conditions. Selective Serotonin Reuptake Inhibitors (SSRIs): Fluoxetine, Sertraline, Citalopram Serotonin-Norepinephrine Reuptake Inhibitors (SNRIs): Venlafaxine, Duloxetine Tricyclic Antidepressants (TCAs): Amitriptyline, Imipramine Monoamine Oxidase Inhibitors (MAOIs): Phenelzine, Selegiline Atypical Antidepressants: Bupropion, Mirtazapine 2. Antipsychotics Used to manage schizophrenia, bipolar disorder, and psychotic disorders. Typical (First-Generation) Antipsychotics: Haloperidol, Chlorpromazine Atypical (Second-Generation) Antipsychotics: Risperidone, Olanzapine, Clozapine 3. Mood Stabilizers Used to treat bipolar disorder and mood dysregulation. Lithium: Effective for bipolar disorder Anticonvulsants: Valproate, Lamotrigine, Carbamazepine 4. Anxiolytics and Sedative-Hypnotics Used for anxiety and sleep disorders. Benzodiazepines: Diazepam, Lorazepam, Alprazolam Non-Benzodiazepine Hypnotics: Zolpidem, Eszopiclone Barbiturates: Phenobarbital (rarely used) Beta-Blockers: Propranolol

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VENTILATORS.pptx FOR NURSING STUDENTS CREATED BY KIRAN KARETHAKIRAN KARETHA

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VENTILATOR Mechanical ventilation is a form of artificial respiration that uses a breathing machine to assist patients with breathing. Mechanical ventilation is the use of a mechanical device to inflate and deflate the lungs. A mechanical ventilation is a machine that generates a controlled flow of gas into a patient’s airways. INDICATIONS Acute lung injury Apnea with respiratory arrest COPD Acute respiratory acidosis Hypoxemia hypercapnia Classification of ventilators: 1. negative pressure ventilators: mechanical ventilators exert a negative pressure on the external chest. Decreasing the intrathoracic pressure during inspiration allows air to flow into the lung, filling its volume. NEGATIVE PRESSURE VENTILATORS 1. Iron lung (drinker respirator tank) 2. body wrap and chest cuirass (tortoise shell) POSITIVE PRESSURE VENTILATORS 1. pressure cycled ventilators 2. time cycled ventilators 3. volume cycled ventilators 4. non-invasive positive pressure ventilators a) continuous positive airway pressure b) bilevel positive airway pressure

antiherpes acyclovir mcq ANKUSH GOYAL GMC PATIALA.docxDr Ankush goyal

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### **Acyclovir and Anti-Herpes Medications** **Acyclovir** is an antiviral medication used to treat infections caused by herpes viruses, including: - **Herpes simplex virus (HSV-1 & HSV-2)** – Causes cold sores and genital herpes. - **Varicella-zoster virus (VZV)** – Causes chickenpox and shingles. ### **Mechanism of Action:** Acyclovir works by inhibiting viral DNA replication. It is activated inside virus-infected cells, where it blocks viral enzymes, preventing the virus from multiplying. ### **Forms & Dosage:** - **Oral (Tablets/Capsules/Liquid)** – Used for mild to moderate infections. - **Topical (Cream/Ointment)** – For cold sores and mild skin infections. - **Intravenous (IV)** – For severe infections like herpes encephalitis. ### **Other Anti-Herpes Medications:** - **Valacyclovir (Valtrex)** – A prodrug of acyclovir with better absorption. - **Famciclovir (Famvir)** – Another antiviral with a longer duration of action. - **Penciclovir (Denavir)** – Used topically for herpes labialis (cold sores). ### **Uses:** - Treats and manages outbreaks of herpes simplex. - Reduces the severity and duration of symptoms. - Used for long-term suppression to prevent recurrent infections. - Helps in managing shingles and chickenpox. ### **Side Effects:** - Nausea, vomiting, diarrhea. - Headache, dizziness, fatigue. - Rare: Kidney issues, allergic reactions. ### **Precautions:** - Drink plenty of fluids to prevent kidney issues. - Not a cure, but helps manage symptoms. - Safe for most patients but should be used cautiously in people with kidney disease or weakened immune systems.

ECG-Interpretation-and-Management-of-Arrhythmias.pptx Dr ankush goyal by Dr Ankush goyal, has 8 slides with 22 views.ECG Interpretation and Management Introduction Electrocardiography (ECG) is a crucial diagnostic tool used to assess the electrical activity of the heart. It provides essential information about heart rate, rhythm, conduction abnormalities, myocardial ischemia, and electrolyte disturbances. Correct interpretation of an ECG requires a systematic approach and understanding of normal and pathological waveforms. Basics of ECG Interpretation 1. ECG Waves and Intervals P wave: Represents atrial depolarization. PR interval: Time from atrial depolarization to ventricular depolarization (normal: 120-200 ms). QRS complex: Ventricular depolarization (normal: <120 ms). ST segment: Represents the interval between ventricular depolarization and repolarization. T wave: Represents ventricular repolarization. QT interval: Duration of ventricular depolarization and repolarization (normal: <450 ms in males, <460 ms in females). 2. Systematic Approach to ECG Interpretation 1. Determine heart rate Regular rhythm: 300 divided by the number of large squares between R waves. Irregular rhythm: Count QRS complexes in 6 seconds and multiply by 10. 2. Assess heart rhythm Regular or irregular? Presence of P waves? Relationship between P waves and QRS complexes? 3. Evaluate cardiac axis Normal: -30 to +90 degrees. Left axis deviation: <-30 degrees (e.g., left anterior hemiblock, left ventricular hypertrophy). Right axis deviation: >+90 degrees (e.g., right ventricular hypertrophy, pulmonary embolism). 4. Analyze P wave morphology Peaked P waves (right atrial enlargement). Broad P waves (left atrial enlargement). 5. Assess PR interval Short PR: Wolff-Parkinson-White syndrome. Prolonged PR: First-degree AV block. 6. Inspect QRS complex Narrow QRS (<120 ms): Normal conduction. Wide QRS (>120 ms): Bundle branch block or ventricular origin. 7. Evaluate ST segment and T waves ST elevation: Myocardial infarction. ST depression: Ischemia or hypokalemia. Inverted T waves: Ischemia, infarction, or hypertrophy. 8. Check QT interval Prolonged QT: Risk of Torsades de Pointes. Short QT: Hypercalcemia. Common ECG Abnormalities and Management 1. Arrhythmias a) Sinus Bradycardia ECG Findings: HR < 60 bpm, normal P waves, and QRS complexes. Causes: Increased vagal tone, hypothyroidism, beta-blockers. Management: Treat underlying cause; consider atropine if symptomatic. b) Sinus Tachycardia ECG Findings: HR > 100 bpm, normal P waves, and QRS complexes. Causes: Fever, dehydration, anemia, hyperthyroidism. Management: Address underlying cause; beta-blockers if needed. c) Atrial Fibrillation ECG Findings: Irregularly irregular rhythm, absent P waves, fibrillatory waves. Causes: Hypertension, valvular heart disease, hyperthyroidism. Management: Rate control (beta-blockers, calcium channel blockers), rhythm control (amiodarone, cardioversion), anticoagulation (warfarin, DOACs). d) Atrial Flutter ECG Findings: Sawtooth flutter

ECG-Interpretation-and-Management-of-Arrhythmias.pptx Dr ankush goyalDr Ankush goyal

8 slides•22 views

ECG Interpretation and Management Introduction Electrocardiography (ECG) is a crucial diagnostic tool used to assess the electrical activity of the heart. It provides essential information about heart rate, rhythm, conduction abnormalities, myocardial ischemia, and electrolyte disturbances. Correct interpretation of an ECG requires a systematic approach and understanding of normal and pathological waveforms. Basics of ECG Interpretation 1. ECG Waves and Intervals P wave: Represents atrial depolarization. PR interval: Time from atrial depolarization to ventricular depolarization (normal: 120-200 ms). QRS complex: Ventricular depolarization (normal: <120 ms). ST segment: Represents the interval between ventricular depolarization and repolarization. T wave: Represents ventricular repolarization. QT interval: Duration of ventricular depolarization and repolarization (normal: <450 ms in males, <460 ms in females). 2. Systematic Approach to ECG Interpretation 1. Determine heart rate Regular rhythm: 300 divided by the number of large squares between R waves. Irregular rhythm: Count QRS complexes in 6 seconds and multiply by 10. 2. Assess heart rhythm Regular or irregular? Presence of P waves? Relationship between P waves and QRS complexes? 3. Evaluate cardiac axis Normal: -30 to +90 degrees. Left axis deviation: <-30 degrees (e.g., left anterior hemiblock, left ventricular hypertrophy). Right axis deviation: >+90 degrees (e.g., right ventricular hypertrophy, pulmonary embolism). 4. Analyze P wave morphology Peaked P waves (right atrial enlargement). Broad P waves (left atrial enlargement). 5. Assess PR interval Short PR: Wolff-Parkinson-White syndrome. Prolonged PR: First-degree AV block. 6. Inspect QRS complex Narrow QRS (<120 ms): Normal conduction. Wide QRS (>120 ms): Bundle branch block or ventricular origin. 7. Evaluate ST segment and T waves ST elevation: Myocardial infarction. ST depression: Ischemia or hypokalemia. Inverted T waves: Ischemia, infarction, or hypertrophy. 8. Check QT interval Prolonged QT: Risk of Torsades de Pointes. Short QT: Hypercalcemia. Common ECG Abnormalities and Management 1. Arrhythmias a) Sinus Bradycardia ECG Findings: HR < 60 bpm, normal P waves, and QRS complexes. Causes: Increased vagal tone, hypothyroidism, beta-blockers. Management: Treat underlying cause; consider atropine if symptomatic. b) Sinus Tachycardia ECG Findings: HR > 100 bpm, normal P waves, and QRS complexes. Causes: Fever, dehydration, anemia, hyperthyroidism. Management: Address underlying cause; beta-blockers if needed. c) Atrial Fibrillation ECG Findings: Irregularly irregular rhythm, absent P waves, fibrillatory waves. Causes: Hypertension, valvular heart disease, hyperthyroidism. Management: Rate control (beta-blockers, calcium channel blockers), rhythm control (amiodarone, cardioversion), anticoagulation (warfarin, DOACs). d) Atrial Flutter ECG Findings: Sawtooth flutter

Artificial Intelligence, Data and Competition – SCHREPEL – June 2024 OECD dis...OECD Directorate for Financial and Enterprise Affairs

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This presentation by Thibault Schrepel, Associate Professor of Law at Vrije Universiteit Amsterdam University, was made during the discussion “Artificial Intelligence, Data and Competition” held at the 143rd meeting of the OECD Competition Committee on 12 June 2024. More papers and presentations on the topic can be found at oe.cd/aicomp. This presentation was uploaded with the author’s consent.

How to Leverage AI to Boost Employee Wellness - Lydia Di Francesco - SocialHR...SocialHRCamp

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Speaker: Lydia Di Francesco In this workshop, participants will delve into the realm of AI and its profound potential to revolutionize employee wellness initiatives. From stress management to fostering work-life harmony, AI offers a myriad of innovative tools and strategies that can significantly enhance the wellbeing of employees in any organization. Attendees will learn how to effectively leverage AI technologies to cultivate a healthier, happier, and more productive workforce. Whether it's utilizing AI-powered chatbots for mental health support, implementing data analytics to identify internal, systemic risk factors, or deploying personalized wellness apps, this workshop will equip participants with actionable insights and best practices to harness the power of AI for boosting employee wellness. Join us and discover how AI can be a strategic partner towards a culture of wellbeing and resilience in the workplace.

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